I’m having trouble with this fill in the blank. Could you please let me know if I need to change any of my answers. Proto-oncogenes normally function toincreasev cell division. Ifa mutation occurs to a proto-oncogene it may producean abnormal proteinthat leads to the formation ofcancerTumor-suppressor genes normally function todecreasev celldivision. If a mutation occurs to a tumor-suppressor gene there isa loss of controlwhich may lead to the formation ofabnormal tissueResearchers have identifiedmoreproto-oncogenescompared to tumor-suppressor genes.Rasis an example of a proto-oncogene whileCasis an example of a tumor-suppressor gene.bp

Proto - oncogenes normally function to stimulating cell division . If a mutation occurs to a proto -…

Proto-oncogenes normally function to increase v cell division. If a mutation occurs to a proto-oncogene it may produce an abnormal protein that leads to the formation of cancer decrease cell Tumor-suppressor genes normally function to division. If a mutation occurs to a tumor-suppressor gene there is a loss of control V which may lead to the formation of abnormal tissue Researchers have identified more proto-oncogenes compared to tumor-suppressor genes. Ras is an example of a proto-oncogene while Cas is an example of a tumor-suppressor gene.

Proto-oncogenes normally function to increase v cell division. If a mutation occurs to a proto-oncogene it may produce an abnormal protein that leads to the formation of cancer decrease cell Tumor-suppressor genes normally function to division. If a mutation occurs to a tumor-suppressor gene there is a loss of control V which may lead to the formation of abnormal tissue Researchers have identified more proto-oncogenes compared to tumor-suppressor genes. Ras is an example of a proto-oncogene while Cas is an example of a tumor-suppressor gene.

Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter17: Developmental Genetics

Section: Chapter Questions

Problem 8TYU: Which of the following statements about cancer is false? (a) oncogenes arise from mutations in...

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Similar questions

Describe the genetic changes that convert proto-oncogenesinto oncogenes or inactivate tumor-suppressor genes.
With regard to cancer cells, which of the following statementsare true?A. Cancer cells are clonal, which means they are derived from asingle mutant cell.B. To become cancerous, cells usually accumulate multiplegenetic changes that eventually result in uncontrolledgrowth.C. Most cancers are caused by oncogenic viruses.D. Cancer cells have lost the ability to properly regulate celldivision.
A p53 knockout mouse in which both copies of p53 are defectivehas been produced by researchers. This type of mouse appears normalat birth. However, it is highly sensitive to UV light. Based onyour knowledge of p53, explain the normal appearance at birth andthe high sensitivity to UV light.
A research study indicated that an agent in cigarette smokecaused the silencing of a tumor-suppressor gene called p53.However, using sequencing, no mutation was found in the DNAsequence for this gene. Give two possible explanations for theseresults.
''In the cellular regulatory pathways that controlcell growth and proliferation, the products of oncogenesare stimulatory components and the products of tumorsuppressor genes are inhibitory components.'' 'Is this statement true? Explain why or why not.
Retinoblastoma is a rare cancer in which tumors developin the retina of the eye. The tumors arise because of theloss of the Rb gene, which codes for a tumor suppressor.Hereditary retinoblastoma usually appears in children whohave inherited only one functional copy of Rb. Explain whythe nonhereditary form of retinoblastoma usually occurs laterin life.
Our government has finite funds to devote to cancer research.Discuss which of the following areas of research you think shouldreceive the most funding.A. Identifying and characterizing oncogenes and tumorsuppressorgenesB. Identifying agents in our environment that cause cancerC. Identifying viruses that cause cancer D. Devising methods aimed at killing cancer cells in the bodyE. Informing the public of the risks involved in exposure tocarcinogensIn the long run, in which of these areas would you expect successfulresearch to be the most effective in decreasing human mortalitydue to cancer?
Cancer-promoting mutations are likely to have different effects on the activity of proteins encoded byproto-oncogenes than they do on proteins encodedby tumor-suppressor genes. Explain.
Thousands of caspase substrateshave been identified. Which onesare the critical proteins that mustbe cleaved to trigger the majorcell remodeling events underlyingapoptosis?
Which of the following statements about cancer is false? (a) oncogenes arise from mutations in proto-oncogenes (b) tumor suppressor genes normally interact with growth-inhibiting factors to block cell division (c) more than 120 cancer-driving genes have been discovered (d) oncogenes were first discovered in mouse models for cancer (e) the development of cancer is usually a multistep process involving both oncogenes and mutated tumor suppressor genes
One important role of Fas and Fas ligand is to medi-ate the elimination of tumor cells by killer lymphocytes.In a study of 35 primary lung and colon tumors, half thetumors were found to have amplified and overexpressed agene for a secreted protein that binds to Fas ligand. How doyou suppose that overexpression of this protein might con-tribute to the survival of these tumor cells? Explain yourreasoning.
Which one of the following events is unlikely to beassociated with cancer?a. mutation of a cellular proto-oncogene in a normaldiploid cellb. a chromosomal translocation with a breakpointnear a cellular proto-oncogenec. deletion of a cellular proto-oncogened. mitotic nondisjunction in a cell carrying a deletionof a tumor-suppressor genee. incorporation of a cellular oncogene into a retroviruschromosome